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Laudenslager's entire reason for existing seems to be to abuse animals for profit. His pretending that these worthless and abusive experiments are somehow to better mankind is ludicrous and painfully transparent. The only way to make a man without a conscience in an industry without a conscience stop his psychotic experiments on helpless animals is to have the very people who give him money to do these things take it away. That's you and me in our tax dollars and the CU students in their bloated, exceedingly high tuition costs. Please stand up and make this stop.


Click here for QUOTES and CRITIQUES FROM PROFESSIONALS regarding Laudenslager's "new" experiments and why they should be stopped

 

November, 2004

A NEW EXPERIMENT INVOLVING THE MONKEYS AT CU IS NOW UNDERWAY


Although we previously reported that none of the CU 34 monkeys was being used in experiments, it was recently learned that Mark Laudenslager has begun yet another questionable project. He will be studying mother monkeys and their babies to determine which mothers give the best care. The babies will later be presented with alcohol to determine if those with less attentive mothers are prone to ingest more alcohol.

Laudenslager's abstract states, "Differences in early maternal care are suggested as one basis for differences in relative risk for consuming higher quantities of ethanol. . . We hypothesize that monkeys experiencing poor quality maternal care during development will demonstrate increased risk for the _expression of aggressive and impulsive behavior patterns as adolescents. We hypothesize that these behavior patterns will be present in monkeys that voluntarily consume greater quantities of ethanol."

Questions that immediately come to mind:

Can bonnet macaque monkeys in the controlled, unnatural environment of a cage be compared to adolescent humans who are subjected to a myriad of varying circumstances in their lives, including peer pressure and other social pressures to drink?

How will this save human lives?

Why are clinical studies involving humans not conducted to get realistic statistics?

Why was this experiment approved by the University of Colorado and funded by the National Institutes of Health?

The entire abstract of this 5-year experiment follows:


Project Title: Early experience and low 5-HT markers in alcohol abuseAbstract: DESCRIPTION (provided by applicant): Adolescents of middle school and high school age are at high risk for alcohol consumption. Nonhuman primates living in social groups provide an excellent model for the study of social influences on biobehavioral development in general and alcohol abuse problem more specifically. The National Institute on Alcohol Abuse and Alcoholism (NIAAA) is interested in encouraging investigators with expertise in primate developmental biology and behavior to seek collaborations with established alcohol researchers to elucidate the neurobiological mechanisms of adolescent alcohol abuse and alcoholism. Herein we submit a revision of a model of biobehavioral development that focuses on the origins of individual differences in voluntary alcohol consumption in young socially housed bonnet macaque monkeys. Differences in early maternal care are suggested as one basis for differences in relative risk for consuming higher quantities of ethanol. Biomarkers of the activity of the serotonergic system (CSF 5HIAA and the prolactin response to fenfluramine) will be evaluated. Subjects will be selected on the basis of a polymorphism in the promoter region of the serotonin transporter protein gene that modulates serotonergic activity. Maternal care will be determined through observation of mother infant interactions during early development in social group reared macaque monkeys. We hypothesize that monkeys experiencing poor quality maternal care during development will demonstrate increased risk for the _expression of aggressive and impulsive behavior patterns as adolescents. We hypothesize that these behavior patterns will be present in monkeys that voluntarily consume greater quantities of ethanol. Low serotonin will have an additive effect with low quality maternal care on levels of aggression, impulsivity, and alcohol consumption. We predict that adolescent monkeys evidencing higher aggressive and impulsive behavior patterns and low serotonin will show 1) increased rates of ethanol consumption under social conditions, 2) a greater increase in ethanol intake in response to a stressor, 3) higher probability that ethanol consumption will be associated with increased aggression when housed socially, and 4) attenuated soporific effects. Finally, involvement of the HPA axis in these relationships will be investigated as an exploratory goal.